A framework for advancing our understanding of cancer-associated fibroblasts

Концептуальная основа для углубления понимания фибробластов, ассоциированных с опухолью
Fiona M. Watt, Zena Werb, Rakesh K. Jain, Tony Hunter, Douglas T. Fearon, Mikala Egeblad, Richard O. Hynes, Alec C. Kimmelman, Mikhail G. Kolonin, Valerie M. Weaver, David G. DeNardo, Ronald M. Evans, Erik Sahai, Florian R. Greten, Thea D. Tlsty, David A. Tuveson, Ashani T. Weeraratna, Ellen Puré, Mara H. Sherman, Sheila A. Stewart, Sunil R. Hingorani, Robert G. Maki, Scott Powers, Igor Astsaturov, Ruth Scherz‐Shouval, Claus Jørgensen, Edna Cukierman, Tobias Janowitz, Daniel C. Ramirez
2020-01-24

CAF heterogeneityCAF-targeted therapycancer-associated fibroblastsmatrix remodellingtumour microenvironment
Cancer-associated fibroblasts (CAFs) are a key component of the tumour microenvironment with diverse functions, including matrix deposition and remodelling, extensive reciprocal signalling interactions with cancer cells and crosstalk with infiltrating leukocytes. As such, they are a potential target for optimizing therapeutic strategies against cancer. However, many challenges are present in ongoing attempts to modulate CAFs for therapeutic benefit. These include limitations in our understanding of the origin of CAFs and heterogeneity in CAF function, with it being desirable to retain some antitumorigenic functions. On the basis of a meeting of experts in the field of CAF biology, we summarize in this Consensus Statement our current knowledge and present a framework for advancing our understanding of this critical cell type within the tumour microenvironment.
1
CAFs represent potential therapeutic targets for improving cancer treatment because of their broad influence on tumour biology.
2
Cancer-associated fibroblasts are key tumour-microenvironment components that deposit and remodel extracellular matrix and engage in reciprocal signalling with cancer cells and infiltrating leukocytes.
3
Effective CAF-targeted strategies should preserve CAF functions that inhibit tumour growth rather than eliminate CAF activity indiscriminately.
4
The consensus statement establishes a framework for advancing understanding of CAF biology and guiding therapeutic development.
5
Therapeutic modulation of CAFs is complicated by limited knowledge of their cellular origins and substantial functional heterogeneity.

cancer-associated fibroblasts (CAFs) within the tumour microenvironment

the origins, functional heterogeneity, matrix-remodelling functions, reciprocal signalling with cancer cells, crosstalk with infiltrating leukocytes, and therapeutic modulation of CAFs

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2020-01-24
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Authors
Fiona M. Watt
Zena Werb
Rakesh K. Jain
Tony Hunter
Douglas T. Fearon
Mikala Egeblad
Richard O. Hynes
Alec C. Kimmelman
Mikhail G. Kolonin
Valerie M. Weaver
David G. DeNardo
Ronald M. Evans
Erik Sahai
Florian R. Greten
Thea D. Tlsty
David A. Tuveson
Ashani T. Weeraratna
Ellen Puré
Mara H. Sherman
Sheila A. Stewart
Sunil R. Hingorani
Robert G. Maki
Scott Powers
Igor Astsaturov
Ruth Scherz‐Shouval
Claus Jørgensen
Edna Cukierman
Tobias Janowitz
Daniel C. Ramirez
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