An Atypical Presentation of Anton Syndrome in a Patient with Preserved Cognition Despite Multiple Cerebral Infarcts: A Case Report

Атипичное проявление синдрома Антона у пациента с сохранными когнитивными функциями несмотря на множественные церебральные инфаркты: клинический случай
Glen P. Davis, R. Andrew Sewell, Boaz Levy, Bruce H. Price, Miles G. Cunningham
2009-01-01

Anton syndromeadvanced glaucomabilateral occipital infarctscortical blindnessvisual anosognosia
Visual anosognosia (Anton syndrome) is a rare complication of cortical blindness that results from injury to the visual association cortex, in which patients who are unable to see deny that they are blind. They sometimes confabulate explanations for their visual problems (“there is not enough light to see”), or endanger themselves in efforts to “prove” that they can see (eg, tripping or walking into walls). Without functioning visual-association centers, these patients lack the concept of sight and are unable to acknowledge their impairment. Usually, Anton syndrome is encountered in patients with bilateral occipital infarcts but no other cause for impaired vision. We present an atypical case of Anton syndrome in which the patient's underlying visual deficit was due to advanced glaucoma.
1
Anton syndrome is a rare form of visual anosognosia in which patients with cortical blindness deny their blindness and may confabulate explanations for visual loss.
2
The case indicates that Anton syndrome can occur when severe preexisting ocular disease, rather than isolated bilateral occipital injury, accounts for the visual deficit.
3
The reported patient represents an atypical Anton syndrome presentation with preserved cognition despite multiple cerebral infarcts.
4
Unlike the usual presentation involving bilateral occipital infarcts as the sole cause of visual impairment, this patient’s underlying visual deficit was due to advanced glaucoma.

Anton syndrome in a patient with advanced glaucoma and multiple cerebral infarcts

Visual anosognosia and preserved cognition despite cortical blindness and multiple cerebral infarcts

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2009-01-01
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Authors
Glen P. Davis
R. Andrew Sewell
Boaz Levy
Bruce H. Price
Miles G. Cunningham
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