Residual cardiovascular risk beyond low-density lipoprotein cholesterol: inflammation, remnant cholesterol, and lipoprotein(a)

Остаточный сердечно-сосудистый риск помимо холестерина липопротеинов низкой плотности: воспаление, остаточный холестерин и липопротеин(a)
Børge G. Nordestgaard, Anders Berg Wulff
2025-04-07
SCID:  54.1/spxgwn62 Discuss with AI
Lp(a)inflammationlipoprotein(a)low-density lipoprotein cholesterol (LDL-C)remnant cholesterolresidual cardiovascular risk
Abstract (AI)
This editorial refers to ‘Residual cholesterol and inflammatory risk in statin-treated patients undergoing percutaneous coronary intervention’, by B. Bay e
Key Findings
1
Addressing inflammation, remnant cholesterol, and lipoprotein(a) is necessary to further reduce cardiovascular events beyond LDL-C control.
2
Elevated lipoprotein(a) is a distinct, LDL-C–independent driver of residual cardiovascular risk.
3
Inflammation contributes to residual cardiovascular risk and represents a modifiable target beyond lipid lowering.
4
Remnant cholesterol is identified as a contributor to residual cardiovascular risk independent of LDL-C.
5
Residual cardiovascular risk persists despite lowering LDL-C, driven by factors beyond LDL-C reduction.
Research Object

Residual cardiovascular risk beyond low-density lipoprotein cholesterol

Research Subject

Contributions of inflammation, remnant cholesterol, and lipoprotein(a) to residual cardiovascular risk after LDL-C lowering

Publication Details
Publication Date
2025-04-07
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Cited by
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Author Information
Authors
Børge G. Nordestgaard
Anders Berg Wulff
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