Hemifacial Spasm: A Neurosurgical Perspective
Гемифациальный спазм: нейрохирургический аспект
2007-01-01
SCID: 54.1/xu42nqxc
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brainstem auditory evoked potentialsdifferential diagnosisfacial nerve compressionhemifacial spasmmicrovascular decompression
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Abstract (AI)
Hemifacial spasm (HFS) is characterized by tonic clonic contractions of the muscles innervated by the ipsilateral facial nerve. Compression of the facial nerve by an ectatic vessel is widely recognized as the most common underlying etiology. HFS needs to be differentiated from other causes of facial spasms, such as facial tic, ocular myokymia, and blepharospasm. To understand the overall craniofacial abnormalities and to perform the optimal surgical procedures for HFS, we are to review the prevalence, pathophysiology, differential diagnosis, details of each treatment modality, usefulness of brainstem auditory evoked potentials monitoring, debates on the facial EMG, clinical course, and complications from the literature published from 1995 to the present time.
Key Findings
1
Compression of the facial nerve by an ectatic blood vessel is identified as the most common underlying cause of hemifacial spasm.
2
Hemifacial spasm consists of tonic-clonic contractions in muscles innervated by the ipsilateral facial nerve.
3
Hemifacial spasm must be distinguished from facial tic, ocular myokymia, and blepharospasm during diagnosis.
4
The article synthesizes literature from 1995 onward to inform optimal neurosurgical management of hemifacial spasm.
5
The review evaluates treatment modalities, surgical considerations, brainstem auditory evoked potential monitoring, facial electromyography, clinical course, and complications.
Research Object
hemifacial spasm (HFS)
Research Subject
its pathophysiology, differential diagnosis, treatment modalities, neuromonitoring, clinical course, and complications
Publication Details
Publication Date
2007-01-01
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