Neuropathology and virus in brain of SARS-CoV-2 infected non-human primates

Нейропатология и вирус в головном мозге инфицированных SARS-CoV-2 приматов, не относящихся к человеку
Bo Ning, Jay Rappaport, Nicholas J. Maness, Robert V. Blair, Ye Hu, Tracy Fischer, Meredith G. Mayer, Ibolya Rutkai, Linh Hellmers, Zhen Huang, Christopher Monjure, Carol Coyne, Rachel Silvestri, Nadia Golden, Krystle Hensley, Kristin Chandler, Gabrielle Lehmicke, Gregory Bix, Kasi Russell‐Lodrigue, Chad J. Roy, Rudolf P. Bohm, Lara Doyle‐Meyers
2022-04-01

SARS-CoV-2 neuropathogenesisbrain endothelial cellshypoxic-ischemic brain injuryneuroinflammationnon-human primate model
Neurological manifestations are a significant complication of coronavirus disease (COVID-19), but underlying mechanisms aren't well understood. The development of animal models that recapitulate the neuropathological findings of autopsied brain tissue from patients who died from severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection are critical for elucidating the neuropathogenesis of infection and disease. Here, we show neuroinflammation, microhemorrhages, brain hypoxia, and neuropathology that is consistent with hypoxic-ischemic injury in SARS-CoV-2 infected non-human primates (NHPs), including evidence of neuron degeneration and apoptosis. Importantly, this is seen among infected animals that do not develop severe respiratory disease, which may provide insight into neurological symptoms associated with "long COVID". Sparse virus is detected in brain endothelial cells but does not associate with the severity of central nervous system (CNS) injury. We anticipate our findings will advance our current understanding of the neuropathogenesis of SARS-CoV-2 infection and demonstrate SARS-CoV-2 infected NHPs are a highly relevant animal model for investigating COVID-19 neuropathogenesis among human subjects.
1
Infected primates show neuron degeneration and apoptosis, even without severe respiratory disease.
2
Neuropathological injury occurs in animals with mild respiratory disease, potentially modeling neurological manifestations of long COVID.
3
SARS-CoV-2-infected non-human primates develop neuroinflammation, microhemorrhages, brain hypoxia, and hypoxic-ischemic neuropathology.
4
SARS-CoV-2-infected non-human primates provide a relevant model for investigating COVID-19 neuropathogenesis.
5
Sparse SARS-CoV-2 is detected in brain endothelial cells, but viral presence does not correlate with CNS injury severity.

Brains and central nervous systems of SARS-CoV-2-infected non-human primates

Neuropathological injury and neuroinflammatory responses, including hypoxic-ischemic damage, microhemorrhages, neuron degeneration, apoptosis, and the distribution and severity of brain infection

Publication Details
Publication Date
2022-04-01
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Authors
Bo Ning
Jay Rappaport
Nicholas J. Maness
Robert V. Blair
Ye Hu
Tracy Fischer
Meredith G. Mayer
Ibolya Rutkai
Linh Hellmers
Zhen Huang
Christopher Monjure
Carol Coyne
Rachel Silvestri
Nadia Golden
Krystle Hensley
Kristin Chandler
Gabrielle Lehmicke
Gregory Bix
Kasi Russell‐Lodrigue
Chad J. Roy
Rudolf P. Bohm
Lara Doyle‐Meyers
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